Bit · Cardio

Acute Coronary Syndromes — STEMI vs NSTEMI vs Unstable Angina

Three points on the same spectrum of coronary thrombosis. The pivot is the troponin and the ECG.

Mechanism

All three result from coronary plaque rupture and thrombosis. The differences come down to (1) how complete the occlusion is and (2) how much myocardium has died:

Differentiator Table

STEMINSTEMIUnstable Angina
OcclusionCompletePartial (or complete + collaterals)Plaque rupture, no necrosis
ECGST elevation (contiguous leads) OR new LBBBST depression / T inversion / normalST depression / T inversion / normal
TroponinELEVATEDELEVATEDNEGATIVE
Infarct depthTransmuralSubendocardialNone (yet)
TreatmentEMERGENT reperfusion (PCI within 90 min)Anti-thrombotic; angiography within 24–72 hAnti-thrombotic; angiography within 24–72 h
MONA-BASHAll ACS: Morphine (if pain), Oxygen if SaO₂<90, Nitrates, Aspirin, β-blocker, ACEi, Statin, HeparinSame

The Pivot

Two questions:

  1. ST elevation on ECG? → STEMI. Call cath lab.
  2. If no ST elevation — is troponin positive? Yes → NSTEMI. No → unstable angina.

Treatment of NSTEMI and UA is identical; only the diagnosis label differs based on troponin.

NBME-Style Stem

A 64-year-old man with hypertension and a 40-pack-year smoking history presents with 2 hours of crushing substernal chest pain radiating to the left jaw. ECG shows 3-mm ST elevation in leads II, III, and aVF. Troponin is pending. Which of the following is the most appropriate next step?
Concept Anchor
All three are coronary plaque rupture with thrombosis. STEMI is complete and the cath lab opens now. NSTEMI is partial with dead cells. Unstable angina is partial without dead cells. Troponin and ECG decide which one.

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