Bit · Endo

DKA vs HHS

Two hyperglycemic emergencies. DKA = insulin is absent, ketones are the headline. HHS = insulin is just low enough to stop fat breakdown but not enough to control glucose — so glucose runs to 1000+ and the patient dries out.

Mechanism

Both start with too little insulin and too much glucose. The pivot is whether there is enough insulin to suppress lipolysis:

Differentiator Table

DKAHHS
Typical patientT1DM (or new dx)Older T2DM
Glucose400–800 mg/dL>600, often >1000
Ketones↑↑ (β-hydroxybutyrate)Minimal / absent
Arterial pH<7.30≥7.30
Anion gap↑↑Normal or mildly ↑
Serum osm<320 mOsm/kg>320 mOsm/kg
Mental statusVariableStupor / coma (osm-driven)
OnsetHours to a dayDays
First-line txIV fluids → insulin drip → K⁺ replacementIV fluids (huge volume) → insulin drip → K⁺

The Pivot

Two numbers decide it:

  1. pH below 7.30 with ketones? → DKA.
  2. Glucose >600 with serum osm >320 and minimal/no ketones? → HHS.

Patient can have features of both ('mixed') — treat as the more severe of the two.

NBME-Style Stem

A 72-year-old woman with type 2 diabetes is brought in obtunded. She was last seen well 3 days ago. Glucose 1180 mg/dL, Na⁺ 152 mEq/L (corrected), bicarbonate 22 mEq/L, arterial pH 7.34, ketones trace, serum osm 348 mOsm/kg. Which of the following is the most appropriate initial step?
Concept Anchor
DKA and HHS are the same disease at different insulin levels — drop insulin to zero and you light up the ketone factory (DKA); leave a trickle of insulin and you suppress ketones but glucose runs the patient dry over days (HHS).

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