Bit · Biochem

Glycogen Storage Diseases (von Gierke, Pompe, Cori, McArdle)

Four classic glycogen storage diseases. Each is a different broken enzyme — and the clinical picture (liver vs heart vs muscle) tells you which one.

Mechanism

Glycogen is the body's short-term glucose reserve. Each disease breaks a different enzyme in the synthesis/breakdown pathway:

Differentiator Table

TypeNameEnzymeHallmark
IVon GierkeGlucose-6-phosphataseSevere fasting hypoglycemia, lactic acidosis, hepatomegaly, doll facies
IIPompeLysosomal α-1,4-glucosidaseCardiomegaly + hypotonia + early death ('Pompe trashes the Pump')
IIICoriDebranching enzymeMilder von Gierke; NORMAL lactate; limit dextrins
VMcArdleMuscle glycogen phosphorylaseExercise intolerance, cramps, myoglobinuria, second-wind phenomenon

The Pivot

Three questions:

  1. Cardiomegaly + hypotonia in infant? → Pompe.
  2. Fasting hypoglycemia + lactic acidosis + hepatomegaly? → von Gierke (lactic acidosis) or Cori (normal lactate).
  3. Exercise-induced cramps with red urine? → McArdle.

NBME-Style Stem

A 9-month-old boy is brought in for severe hypotonia and failure to thrive. Examination shows a macroglossia and massive cardiomegaly on chest x-ray. Echocardiography shows a thickened, hypocontractile left ventricle. Skin fibroblast assay reveals deficiency of acid α-1,4-glucosidase. Which of the following is the most likely diagnosis?
Concept Anchor
Four storage diseases, four broken steps, four organs: liver fails in von Gierke and Cori, heart fails in Pompe, muscle fails in McArdle. Match the organ to the enzyme.

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