Bit · Heme/Onc

Hemophilia A vs B vs vWD

Three bleeding disorders that share the same chief complaint — easy bruising and joint bleeds — but split on PTT, factor levels, and inheritance. The pivot is usually the bleeding pattern (mucocutaneous vs deep).

Mechanism

All three are problems in the clotting cascade or its launchpad:

Differentiator Table

Hemophilia AHemophilia BvWD
InheritanceX-linked recessiveX-linked recessiveAutosomal dominant (mostly)
Factor missingVIIIIXvWF (and ↓ VIII secondarily)
PTNormalNormalNormal
PTTNormal or mildly ↑
Bleeding time / PFANormalNormal
Bleeding patternDeep — joints, muscleDeep — joints, muscleMucocutaneous — epistaxis, menorrhagia, gums
Sex affectedMalesMalesBoth
First-line txRecombinant factor VIII (or DDAVP for mild)Recombinant factor IXDDAVP (releases stored vWF)

The Pivot

Two questions decide it:

  1. Is the bleeding deep (joint/muscle) or mucocutaneous (nose/gums/menses)? Deep → hemophilia. Mucocutaneous → vWD.
  2. If hemophilia, A or B? Indistinguishable clinically. NBME will give you a factor assay.

Ristocetin cofactor assay is abnormal in vWD — it's the test that proves platelets can't bind vWF.

NBME-Style Stem

A 14-year-old girl is evaluated for heavy menstrual bleeding lasting 9 days and recurrent epistaxis. Her mother and maternal grandmother had similar symptoms. PT is normal, PTT is mildly prolonged, platelet count is normal. Bleeding time is prolonged. Which of the following is the most likely diagnosis?
Concept Anchor
vWF is both a platelet glue and a factor VIII bodyguard — lose it and you bleed like a platelet patient and mildly like a hemophilia patient at the same time.

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