Bit · Heme/Onc

Myeloproliferative Neoplasms — PV, ET, PMF, CML

Four clonal expansions of mature myeloid cells. Same family, four different cell lines run wild. The pivot is which cell line is overproduced and the molecular marker.

Mechanism

Myeloproliferative neoplasms (MPNs) are clonal expansions of mature hematopoietic cells. All originate from a hematopoietic stem cell mutation — three of them share JAK2 V617F mutations to varying degrees, while CML has its own translocation:

Differentiator Table

DiseasePredominant cellDriver mutationHallmarkTreatment
PVRBCsJAK2 V617F (>95%)Aquagenic pruritus, plethora, ↑ Hct, thrombosisPhlebotomy + low-dose aspirin; hydroxyurea if high-risk
ETPlateletsJAK2 / CALR / MPLVery high platelets, thrombosis OR bleedingAspirin; hydroxyurea
PMFMarrow fibrosisJAK2 / CALR / MPLTeardrop cells, leukoerythroblastic smear, massive splenomegaly, dry tapRuxolitinib; HSCT in young
CMLMyeloidBCR-ABL t(9;22)Massive WBC at all maturation stagesImatinib

The Pivot

Two questions:

  1. Which cell line is up? RBC → PV. Platelets → ET. WBC with all stages → CML.
  2. Marrow fibrosis with teardrop cells and leukoerythroblastic smear? → PMF.

Molecular marker confirms: BCR-ABL = CML. JAK2 V617F = consistent with PV (or ET/PMF).

NBME-Style Stem

A 62-year-old man presents with itching after hot showers and reddish discoloration of his face. Hemoglobin is 19 g/dL, hematocrit 58%. JAK2 V617F mutation is positive. Erythropoietin level is suppressed. Which of the following is the most likely diagnosis?
Concept Anchor
Each MPN is one mature blood line cloned out of control. The cell that's high in the CBC plus the right driver mutation closes the diagnosis.

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